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Brunden, K.

Alzheimer's disease

Evidence that non-fibrillar tau causes pathology linked to neurodegeneration and behavioral impairments. Alzheimers Dis. Bullido, M. Burdick, D. Busche, M. Tau impairs neural circuits, dominating amyloid-beta effects, in Alzheimer models in vivo. Calderon-Garciduenas, A.

Alzheimer disease. Caldwell, A. Castillo-Carranza, D.

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Passive immunization with Tau oligomer monoclonal antibody reverses tauopathy phenotypes without affecting hyperphosphorylated neurofibrillary tangles. Cataldo, A. Abeta localization in abnormal endosomes: association with earliest abeta elevations in AD and Down syndrome.

Aging 25, — Chen, S. Interaction between human prion protein and amyloid-beta Abeta oligomers: role OF N-terminal residues. Chen, X. Dysregulation of neurotrophin signaling in the pathogenesis of Alzheimer disease and of Alzheimer disease in Down syndrome. Free Radic. Cisse, M.

Alzheimer's disease - Wikipedia

Reversing EphB2 depletion rescues cognitive functions in Alzheimer model. Nature , 47— Cline, E. The amyloid-beta oligomer hypothesis: beginning of the third decade. Corder, E. Science , — Curtis, C. Phase 3 trial of flutemetamol labeled with radioactive fluorine 18 imaging and neuritic plaque density. JAMA Neurol. Histopathology 38, — Davidson, Y. Davis, M. Alzheimer Res. Neuron 73, — De Felice, F. Abeta oligomers induce neuronal oxidative stress through an N-methyl-D-aspartate receptor-dependent mechanism that is blocked by the Alzheimer drug memantine. Deane, R.

RAGE mediates amyloid-beta peptide transport across the blood-brain barrier and accumulation in brain. DeKosky, S. Di Battista, A. Di Paolo, G. DiChiara, T. Yale J. Dohler, F. Doran, E. Dorszewska, J. Dubois, B. Dujardin, S. Neuron-to-neuron wild-type Tau protein transfer through a trans-synaptic mechanism: relevance to sporadic tauopathies. Ellis, W. Neurology 24, — Esparza, T.

Amyloid-beta oligomerization in Alzheimer dementia versus high-pathology controls. Fandos, N. Farrer, L. Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and alzheimer disease meta analysis consortium. JAMA , — Filippi, L. Imaging 8, — Fortea, J. Frost, B. Propagation of tau misfolding from the outside to the inside of a cell.

Funk, K. Galloway, P. Hirano body filaments contain actin and actin-associated proteins. Genin, E. APOE and Alzheimer disease: a major gene with semi-dominant inheritance. Psychiatry 16, — Georganopoulou, D. Giannakopoulos, P.

Neurology 60, — Gimbel, D. Memory impairment in transgenic Alzheimer mice requires cellular prion protein. Goldman, J. The association of actin with Hirano bodies. Gomez-Isla, T. Gouras, G. Intraneuronal Abeta42 accumulation in human brain. Gravina, S. Biochemical and immunocytochemical analysis with antibodies specific for forms ending at A beta 40 or A beta 42 Grundke-Iqbal, I. Abnormal phosphorylation of the microtubule-associated protein tau tau in Alzheimer cytoskeletal pathology. Gyure, K. Intraneuronal abeta-amyloid precedes development of amyloid plaques in Down syndrome.


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Haass, C. Trafficking and proteolytic processing of APP. Cold Spring Harb. Hamos, J. Neurology 39, — Hardy, J. Harris, J. Transsynaptic progression of amyloid-beta-induced neuronal dysfunction within the entorhinal-hippocampal network. Head, E. Cerebrovascular pathology in down syndrome and Alzheimer disease. Hersi, M. Neurotoxicology 61, — Hirano, A. Hirano bodies and related neuronal inclusions.

Hithersay, R.